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eclipse 80i bright field microscope  (Nikon)


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    Structured Review

    Nikon eclipse 80i bright field microscope
    Eclipse 80i Bright Field Microscope, supplied by Nikon, used in various techniques. Bioz Stars score: 99/100, based on 57094 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/bright+field+microscope/pmc12828810-93-12-11?v=Nikon
    Average 99 stars, based on 57094 article reviews
    eclipse 80i bright field microscope - by Bioz Stars, 2026-07
    99/100 stars

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    Image Search Results


    USP18 deficiency inhibits the growth of ccRCC organoids (A and B) Bright-field (A, scale bars, 500 μm) and H&E (B, scale bars, 100 μm) images showing the morphology and structure of ccRCC organoids from three patient samples. (C and D) IF data show the expression of CK7 (C) and Vimentin (D) in ccRCC organoids (scale bars, 100 μm). (E) Bright-field images showing the morphology of control and USP18-deficient ccRCC organoids. “Before” indicates the bright-field image of the organoids after transfection with shNC/shUSP18 at 72 h, and “after” indicates the bright-field image of the organoids after transfection with shNC/shUSP18 at 96 h. The images taken before and after are of the same organoids at different time points in the same field of view. (F) Change in diameter (%) of control and USP18-deficient ccRCC organoids. (G) ATP assay indicating the viability of control and USP18-deficient ccRCC organoids. (H and I) Western blot data indicate the expression of USP18, YBX3, P-AKT, and P-PI3K in control and USP18-deficient ccRCC organoids. Unpaired two-tailed Student’s t test was employed for p -value calculation. Data are represented as mean ± SD.

    Journal: iScience

    Article Title: USP18 promotes clear cell renal cell carcinoma progression by regulating the ubiquitination and stability of YBX3

    doi: 10.1016/j.isci.2026.115808

    Figure Lengend Snippet: USP18 deficiency inhibits the growth of ccRCC organoids (A and B) Bright-field (A, scale bars, 500 μm) and H&E (B, scale bars, 100 μm) images showing the morphology and structure of ccRCC organoids from three patient samples. (C and D) IF data show the expression of CK7 (C) and Vimentin (D) in ccRCC organoids (scale bars, 100 μm). (E) Bright-field images showing the morphology of control and USP18-deficient ccRCC organoids. “Before” indicates the bright-field image of the organoids after transfection with shNC/shUSP18 at 72 h, and “after” indicates the bright-field image of the organoids after transfection with shNC/shUSP18 at 96 h. The images taken before and after are of the same organoids at different time points in the same field of view. (F) Change in diameter (%) of control and USP18-deficient ccRCC organoids. (G) ATP assay indicating the viability of control and USP18-deficient ccRCC organoids. (H and I) Western blot data indicate the expression of USP18, YBX3, P-AKT, and P-PI3K in control and USP18-deficient ccRCC organoids. Unpaired two-tailed Student’s t test was employed for p -value calculation. Data are represented as mean ± SD.

    Article Snippet: Inverted bright-field microscope , Motic , N/A.

    Techniques: Expressing, Control, Transfection, ATP Assay, Western Blot, Two Tailed Test

    USP18 deficiency inhibits the growth of ccRCC organoids (A and B) Bright-field (A, scale bars, 500 μm) and H&E (B, scale bars, 100 μm) images showing the morphology and structure of ccRCC organoids from three patient samples. (C and D) IF data show the expression of CK7 (C) and Vimentin (D) in ccRCC organoids (scale bars, 100 μm). (E) Bright-field images showing the morphology of control and USP18-deficient ccRCC organoids. “Before” indicates the bright-field image of the organoids after transfection with shNC/shUSP18 at 72 h, and “after” indicates the bright-field image of the organoids after transfection with shNC/shUSP18 at 96 h. The images taken before and after are of the same organoids at different time points in the same field of view. (F) Change in diameter (%) of control and USP18-deficient ccRCC organoids. (G) ATP assay indicating the viability of control and USP18-deficient ccRCC organoids. (H and I) Western blot data indicate the expression of USP18, YBX3, P-AKT, and P-PI3K in control and USP18-deficient ccRCC organoids. Unpaired two-tailed Student’s t test was employed for p -value calculation. Data are represented as mean ± SD.

    Journal: iScience

    Article Title: USP18 promotes clear cell renal cell carcinoma progression by regulating the ubiquitination and stability of YBX3

    doi: 10.1016/j.isci.2026.115808

    Figure Lengend Snippet: USP18 deficiency inhibits the growth of ccRCC organoids (A and B) Bright-field (A, scale bars, 500 μm) and H&E (B, scale bars, 100 μm) images showing the morphology and structure of ccRCC organoids from three patient samples. (C and D) IF data show the expression of CK7 (C) and Vimentin (D) in ccRCC organoids (scale bars, 100 μm). (E) Bright-field images showing the morphology of control and USP18-deficient ccRCC organoids. “Before” indicates the bright-field image of the organoids after transfection with shNC/shUSP18 at 72 h, and “after” indicates the bright-field image of the organoids after transfection with shNC/shUSP18 at 96 h. The images taken before and after are of the same organoids at different time points in the same field of view. (F) Change in diameter (%) of control and USP18-deficient ccRCC organoids. (G) ATP assay indicating the viability of control and USP18-deficient ccRCC organoids. (H and I) Western blot data indicate the expression of USP18, YBX3, P-AKT, and P-PI3K in control and USP18-deficient ccRCC organoids. Unpaired two-tailed Student’s t test was employed for p -value calculation. Data are represented as mean ± SD.

    Article Snippet: The morphology of organoids was examined using an inverted bright-field microscope (Motic, China) equipped with a digital camera.

    Techniques: Expressing, Control, Transfection, ATP Assay, Western Blot, Two Tailed Test